IIT Madras IIT Madras IIT Madras IIT Madras
Dept. of Biotechnology, Indian Institute of Technology, Madras,INDIA

 

 

 

 

Dr.Amal kanti bera

PERSONAL

Educational Background

*    PhD.: Biophysics, University of Delhi, India. 1999.

*    M.Sc.: Physiology, University of Calcutta, India. 1991.

*    B.Sc.: Physiology, from City College, University of Calcutta, India. 1989.

 

Career Profile

* Assistant Professor: Department of Biotechnology, Indian Institute of Technology Madras, Chennai-600036 (December 12, 2005-present).

*    Research Associate: Albert Einstein College of Medicine, (October 2000 - November 2005).

*    Post Doctoral Fellow: Department of Physiology, UT Southwestern Medical Center at Dallas, Texas. (January 2000 - September 2000).

*    Post Doctoral Fellow: Department of Physiology and Pharmacology, Sackler School of Medicine, Tel Aviv University, Israel. (1998-1999).

 

Poster Presented in Scientific meeting

XVI International symposium organized by International Union of Biochemistry and Molecular Biology, held in New Delhi, India, 1994.

National symposium on Cellular and Molecular Biophysics, held in Hyderabad, India, 1995.

4th International Symposium on Biochemical Roles of Eukaryotic Cell Surface Macromolecules, held in New Delhi, India, 1996.

National Seminar on Perspectives in Interfacial Areas of Chemistry and Biology, held in New Delhi, India, 1998.

Annual meeting of Society for Neuroscience, held in San Diego, California, 2001.

Annual meeting of Society for Neuroscience, held in Orlando, Florida, 2002.

Annual meeting of Society for Neuroscience, held in New Orleans, Louisiana, 2003.

Annual meeting of Society for Neuroscience, held in San Diego, 2004.

Professional Memberships

Society for Neuroscience, USA

New York Academy of Science, USA

 

Honours and Awards

*    Young Scientist Award, by International Union of Biochemistry and Molecular Biology, to participate in XVI- International Symposium held in New Delhi, India, 1994.

*    Senior Research Fellowship awarded by Council of Scientific and Industrial Research, Government of India, 1995-98.

*    Travel grant, awarded by Council of Scientific and Industrial Research, Government of India, to participate in Biophysical Society meeting held in USA, 1995

Publications

     Bera, A.K., Ghosh, S., Das S. 1995. Mitochondrial VDAC can be phosphorylated by cAMP dependent protein kinase. Biochem. Biophys. Res.Commun. 209(1), 213-217.

     Chandra, A., Ghosh, P., Mandaokar, D., Bera, A.K., Sharma, R.P., Das, S., Kumar, P.A. 1999. Amino acid substitution in helix 7 of Cry1Ac -endotoxin of Bacillus thuringiensis leads to enhanced toxicity of Helicoverpa armigera Hubner. FEBS Letters 458, 175-179.

     Ghosh, S., Bera, A.K., Das, S.1999. Evidence for Nonlinear Capacitance in Biomembrane Channel System. J Theor Biol. 200(3): 299-305.

     Vijayvergiya, V., Ghosh, P., Bera, A.K., and Das, S.1999. Bis[(benzo-15-crown-5)-15-yl-Methyl] pimelate forms ion channels in BLM: A novel model ion channel. Physiol. Chem.Physics & Med. NMR. , 31:93-102.

     Vorobiov, D., Bera A.K., Raifman, T., Barzilai, R. and Dascal, N. 2000. Coupling of the muscarinic m2 receptor to G protein-activated K+ channels via G z and a receptor-G z fusion protein. J. Biol. Chem. 275(6): 4166-70. (*equal contribution with first author).

     Mullner C., Vorobiov D., Bera A.K., Uezono Y, Yakubovich D, Frohnwieser-Steinecker B, Dascal N, Schreibmayer W.2000. Heterologous Facilitation of G Protein-activated K+ Channels By beta-Adrenergic Stimulation via cAMP-dependent Protein Kinase. J Gen Physiol 115(5): 547-558.

     Shin D.M., Zhao X.S., Luo X, Bera A.K., Muallem S. 2000 Specificity of Ca2+ signaling in polarized cells. Adv Dent Res, 14:12-50.

     Ghosh S. and Bera A.K., 2001. The Role of H+ Concentration on the Capacitance of a Membrane Channel Protein. J Theor Biol. 208,383-384.

     Raifman T.K., Bera A.K., Zevig D., Peleg S., Witherow S.D., Slepak V.Z. and Dascal N. 2001. Expression levels of RGS7 and RGS4 proteins determine the mode of regulation of the G protein-activated K (+) channel and control regulation of RGS7 by G beta 5. FEBS Lett. 492(1-2), 20-28.

     Luo X., Popov S., Bera A.K., Wilkie T.M. and Muallem. S. 2001. RGS Proteins Provide Biochemical Control of Agonist-Evoked [Ca2+]i Oscillations. Molecular Cell 7, 651-660.

     Bera A.K., Ghosh S. 2001. Dual mode of gating of voltage-dependent anion channel as revealed by phosphorylation. J Struct Biol, 135(1): 67-72.

     Bera A. K., Chatav M. and Akabas M.H. 2002. GABAA Receptor M2-M3 Loop Secondary Structure and Changes in Accessibility During Channel Gating. J. Biol. Chem. 277(45) 43002-43010.

     Bera , A.K., and Akabas M.H. 2005. Spontaneous thermal motion of the GABAA receptor M2 channel-lining segments. J. Biol. Chem. 280(42):35506-1

 

Book Chapter

  Akabas, M.H., Horenstein, J., Williams, D.B., Bali, M., and Bera, A.K., 2002. GABAA Receptors: Probing the Structural Basis of Function, Gating and Pharmacological Modulation by General Anesthetics. Molecular and Basic Mechanisms of Anesthesia.Ed. M. Barann and B.W. Urban. Pabst Science Publishers, Germany.

 

 

 

 

 

 

 

 

 

 

 

Ion Channel Research Group